BIN1 deficit impairs brain cell communication, memory consolidation

(University of South Florida (USF Health)) A new study has discovered that a lack of neuronal protein BIN1 leads to a defect in the transmission of chemical messages that activate brain cell communication allowing us to think, remember and behave. The preclinical research by a team from the University of South Florida Health (USF Health) Morsani College of Medicine and the University of Chicago provides new insights into how BIN1 may boost Alzheimer’s disease risk.